Cytarabine

證據等級: L5 預測適應症: 9

目錄

  1. Cytarabine
  2. Cytarabine: From Acute Myeloid Leukemia to Small Cell Lung Carcinoma
    1. One-Sentence Summary
    2. Quick Overview
    3. Why is This Prediction Reasonable?
    4. Clinical Trial Evidence
    5. Literature Evidence
    6. Norway Market Information
    7. Cytotoxicity
    8. Safety Considerations
    9. Conclusion and Next Steps
    10. Disclaimer

## 藥師評估報告

Cytarabine: From Acute Myeloid Leukemia to Small Cell Lung Carcinoma

One-Sentence Summary

Cytarabine (Ara-C) is a pyrimidine nucleoside antimetabolite long established as a backbone agent for acute myeloid leukemia and other hematologic malignancies. The TxGNN model predicts it may be effective for Small Cell Lung Carcinoma (SCLC), with 3 clinical trials (none directly testing cytarabine) and 20 publications, largely from the 1970s–1990s, currently supporting this direction.

Quick Overview

Item Content
Original Indication Not recorded in local regulatory data (drug is not marketed locally); globally established for acute myeloid leukemia and other hematologic malignancies
Predicted New Indication Small Cell Lung Carcinoma
TxGNN Prediction Score 99.78%
Evidence Level L3
Norway Market Status 未上市 (Not Marketed)
Number of Authorizations 0
Recommended Decision Hold

Why is This Prediction Reasonable?

Currently, detailed mechanism of action data is not available. Based on known information, cytarabine is a cell-cycle (S-phase) specific pyrimidine nucleoside analog that incorporates into DNA and inhibits DNA polymerase, and its efficacy in acute leukemia has been proven for decades. Mechanistically it has also been explored — mostly in older literature — for solid tumors with rapid proliferation, including lung cancer.

Small cell lung carcinoma is characterized by a very high growth fraction, which in principle makes it a plausible target for an S-phase-specific cytotoxic agent like cytarabine. Historical combination regimens (e.g., cyclophosphamide/doxorubicin/cytosine arabinoside plus radiotherapy, and cytarabine plus cisplatin/vindesine) were tested in NSCLC and SCLC in the late 1970s through 1990s, showing measurable but modest activity, often limited by significant hematologic toxicity.

However, none of the clinical trials currently linked to this prediction directly test cytarabine — they primarily evaluate intrathecal pemetrexed or unrelated NSCLC adjuvant regimens. The supporting mechanistic rationale therefore relies on older cohort/pilot studies rather than modern, disease-specific trial designs, which limits confidence in this prediction.

Clinical Trial Evidence

Trial Number Phase Status Enrollment Key Findings
NCT03507244 Phase 1/2 Completed 34 Intrathecal pemetrexed with concurrent involved-field radiotherapy for leptomeningeal metastasis from solid tumors; does not test cytarabine, disease overlap only
NCT03101579 Phase 1 Completed 13 Intrathecal pemetrexed for recurrent leptomeningeal metastasis from NSCLC; cytarabine mentioned only as a comparator class, not tested
NCT00863512 Phase 3 Terminated 34 Adjuvant chemotherapy (vinorelbine, cisplatin, docetaxel, gemcitabine, pemetrexed) in early-stage NSCLC; cytarabine not part of regimen, trial terminated

Literature Evidence

PMID Year Type Journal Key Findings
2157307 1990 Phase 2 trial Tumori Cytarabine + cisplatin + vindesine in 32 advanced NSCLC patients; 18% response rate
2156598 1990 Phase 2 trial Cancer High-dose cytarabine + cisplatin in 37 chemo-naive NSCLC patients; 14% response rate, Grade III/IV myelosuppression in 46%
2820740 1987 Pilot trial Eur J Cancer Clin Oncol Cisplatin + cytarabine combination pilot study in advanced NSCLC
232239 1979 Cohort/combined modality Med Pediatr Oncol 20 SCLC patients treated with cyclophosphamide/Adriamycin + subcutaneous cytosine arabinoside plus radiotherapy
6095640 1984 Historical trial Am J Clin Oncol Continuous-infusion Ara-C alone (no response, severe toxicity) and Ara-C added to CAV regimen in SCLC
2841844 1988 Historical trial Am J Clin Oncol Etoposide + infusional Ara-C in relapsed/refractory SCLC (17 patients)
348088 1978 Review Antibiot Chemother Review of Ara-C analogs and strategies to prolong biological activity against cytidine deaminase deactivation
9561978 1998 Case series Arch Neurol Combined modality therapy for carcinomatous meningitis secondary to NSCLC
6264785 1981 Case report Am J Med Meningeal carcinomatosis in SCLC patients receiving intensive chemotherapy
28223673 2017 Case report Gan To Kagaku Ryoho Multidisciplinary treatment of meningeal carcinomatosis in SCLC

Norway Market Information

This drug currently has no marketing authorizations recorded in the reviewed regulatory data (market status: 未上市 / Not Marketed, 0 authorizations).

Cytotoxicity

Item Content
Cytotoxicity Classification Conventional cytotoxic (pyrimidine nucleoside antimetabolite / S-phase-specific antileukemic agent)
Myelosuppression Risk High — historical NSCLC combination data report Grade IV myelosuppression in 32% and Grade III in 14% of patients treated with high-dose cytarabine + cisplatin
Emetogenicity Classification Moderate to High (dose-dependent; nausea/vomiting reported as principal non-hematologic toxicity in high-dose regimens)
Monitoring Items CBC with differential, liver and renal function, neurological exam (high-dose neurotoxicity), ocular exam (conjunctivitis risk with high-dose regimens)
Handling Protection Must follow cytotoxic drug handling regulations

Safety Considerations

Please refer to the package insert for safety information.

Conclusion and Next Steps

Decision: Hold

Rationale: A blocking data gap exists — local prescribing warnings/contraindications are unavailable, which prevents entry into S1 safety pre-assessment. In addition, none of the linked clinical trials directly test cytarabine for SCLC, and the supporting evidence is largely historical (1970s–1990s) with modest efficacy and significant myelosuppression; the drug is also not currently marketed locally.

To proceed, the following is needed:

  • Local product label / regulatory safety data (warnings, contraindications, DDI) — currently blocking
  • Detailed mechanism of action (MOA) data from DrugBank
  • Modern, disease-specific clinical evidence directly evaluating cytarabine in SCLC (current linked trials do not test the drug)
  • Route of administration compatibility assessment
  • Local market/import pathway assessment given current "not marketed" status

    Disclaimer

This content is for research purposes only and does not constitute medical advice. Clinical validation is required before any clinical application.



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