Hydrocortisone

證據等級: L5 預測適應症: 10

目錄

  1. Hydrocortisone
  2. Hydrocortisone: From Corticosteroid/Anti-inflammatory Therapy to Alopecia Areata
    1. One-Sentence Summary
    2. Quick Overview
    3. Why is This Prediction Reasonable?
    4. Clinical Trial Evidence
    5. Literature Evidence
    6. Norway Market Information
    7. Safety Considerations
    8. Conclusion and Next Steps
    9. Disclaimer

## 藥師評估報告

Hydrocortisone: From Corticosteroid/Anti-inflammatory Therapy to Alopecia Areata

One-Sentence Summary

Hydrocortisone is a glucocorticoid classically used for adrenocortical insufficiency and as an anti-inflammatory/immunosuppressive agent (specific approved-indication text is not documented in this evidence pack). The TxGNN model predicts it may be effective for Alopecia Areata, with 4 clinical trials (including 1 completed Phase 3 RCT) and 21 publications currently supporting this direction.


Quick Overview

Item Content
Original Indication Not documented in evidence pack (taiwan_regulatory.licenses and drug.original_indications are both empty). General pharmacological class: corticosteroid, used for adrenal insufficiency / inflammatory-immune conditions
Predicted New Indication Alopecia areata
TxGNN Prediction Score 99.97%
Evidence Level L1
Norway Market Status ✗ Not marketed
Number of Authorizations 0
Recommended Decision Proceed with Guardrails

Why is This Prediction Reasonable?

Currently, detailed mechanism of action data is not available (original_moa: Data Gap). Based on general pharmacological knowledge, hydrocortisone is a corticosteroid class agent whose anti-inflammatory and immunosuppressive activity is well established across dermatologic and endocrine indications; specific TFDA/Norway-approved indication text is not present in this evidence pack.

Alopecia areata is a T-cell-mediated autoimmune attack on hair follicles. Topical corticosteroids, including hydrocortisone, are believed to act by suppressing local cytokine release and lymphocytic infiltration, thereby helping to restore the hair follicle's immune privilege. This is an established, widely used mechanism in dermatology rather than a novel hypothesis — it is directly reflected in the completed Phase 3 pediatric RCT (NCT01453686 / PMID 24226568) comparing hydrocortisone 1% cream with clobetasol propionate 0.05% cream, and echoed by decades of older case-series literature describing intradermal/intracutaneous hydrocortisone injection for alopecia areata and alopecia totalis.

Because hydrocortisone's anti-inflammatory mechanism is generic to the corticosteroid class rather than specific to any single original indication, its mechanistic applicability to an inflammatory/autoimmune dermatologic condition such as alopecia areata is biologically plausible even without a clearly recorded "original indication" in this pack.


Clinical Trial Evidence

Trial Number Phase Status Enrollment Key Findings
NCT01453686 Phase 3 Completed 41 RCT directly comparing clobetasol propionate 0.05% cream vs. hydrocortisone 1% cream in children with alopecia areata — highest-relevance trial (Grade A)
NCT00484679 Phase 2 Completed 18 Evaluated adrenal function impact of intralesional triamcinolone (not hydrocortisone) in alopecia areata — indirect relevance
NCT06551818 N/A Not yet recruiting 72 Four-arm dose-response study of hair growth products vs. placebo in androgenic alopecia; corticosteroid arm composition not yet confirmed
NCT04343560 N/A Completed 380 Studied steroid metabolome effects on bone strength/density in adrenal adenoma patients — not directly related to AA treatment efficacy

Literature Evidence

PMID Year Type Journal Key Findings
24226568 2014 RCT JAMA Dermatology Published RCT results: hydrocortisone 1% vs. clobetasol propionate 0.05% for alopecia areata in children
38501938 2024 Cohort/Interventional Clinical and Experimental Dermatology Retrospective analysis of topical corticosteroid under occlusion for severe alopecia areata (including alopecia totalis/universalis) in children
36718837 2023 Review Journal of Cosmetic Dermatology Systematic review/meta-analysis of AA treatment landscape, including corticosteroid comparators
13368875 1956 Case series Medical Times Early case series treating alopecia areata, partialis and totalis with cortisone, hydrocortisone, prednisone and prednisolone
28516731 2017 Review JEADV Reviews HPA-axis/cortisol activity in alopecia areata pathophysiology
5989830 1966 Case series Vestnik Dermatologii i Venerologii Treatment of alopecia areata and total alopecia with intracutaneous hydrocortisone injections
13610145 1958 Case report Der Hautarzt Hair regrowth in alopecia areata/maligna after intracutaneous hydrocortisone injection
14158891 1963 Case report Actas Dermo-Sifiliográficas Treatment of alopecia areata with intradermal hydrocortisone injections
22381765 2012 Mechanistic Journal of Southern Medical University Serum cortisol and glucocorticoid receptor mRNA expression in severe alopecia areata
15692503 2005 Case report Journal of the American Academy of Dermatology Describes 4 cases of congenital alopecia areata, treatments included topical corticosteroids

Norway Market Information

Hydrocortisone currently has no market authorizations on record in this dataset (market_status: 未上市 / Not marketed; total_licenses: 0). No license table is available to display.


Safety Considerations

Please refer to the package insert for safety information. (key_warnings, contraindications, and ddi are all marked as data gaps in this evidence pack; DG001 flags TFDA label warnings/contraindications as a Blocking data gap.)


Conclusion and Next Steps

Decision: Proceed with Guardrails

Rationale: The lead prediction (alopecia areata) is supported by a completed Phase 3 pediatric RCT (NCT01453686/PMID 24226568) directly comparing hydrocortisone with an active corticosteroid comparator, reinforced by decades of case-series literature on hydrocortisone injection for alopecia areata — yielding an L1 evidence level. However, the drug is not currently marketed in Norway, and both mechanism-of-action data and TFDA-equivalent safety labeling (warnings/contraindications) are missing, preventing a full S1 safety assessment.

To proceed, the following is needed:

  • TFDA/Norway package insert warnings and contraindications (DG001 — Blocking; required before initial safety screening)
  • Detailed mechanism-of-action data from DrugBank (DG002)
  • Confirmation of Norway market/registration pathway, since the drug currently has zero authorizations on record
  • A formal drug interaction (DDI) profile, since the current query returned no results
  • Clarification of the specific original approved indication(s), which were not populated in this evidence pack

    Disclaimer

This content is for research purposes only and does not constitute medical advice. Clinical validation is required before any clinical application.



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